Search results for " Temporal lobe epilepsy"

showing 5 items of 5 documents

Are mutations in the dhrs9 gene causally linked to epilepsy? A case report

2020

The DHRS9 gene is involved in several pathways including the synthesis of allopregnanolone from progesterone. Allopregnanolone is a positive modulator of gamma aminobutyric acid (GABA) action and plays a role in the control of neuronal excitability and seizures. Whole-exome sequencing performed on a girl with an early onset epilepsy revealed that she was a compound heterozygote for two novel missense mutations of the DHRS9 gene likely to disrupt protein function. No previous studies have reported the implication of this gene in epilepsy. We discuss a new potential pathogenic mechanism underlying epilepsy in a child, due to a defective progesterone pathway.

0301 basic medicineCase ReportCompound heterozygosityBioinformaticsAllopregnanolone DHRS9 Exome GABA NGS Temporal lobe epilepsygamma-Aminobutyric acid03 medical and health scienceschemistry.chemical_compoundEpilepsyGABA0302 clinical medicinemedicineMissense mutationGeneExomelcsh:R5-920business.industryMechanism (biology)DHRS9AllopregnanoloneallopregnanoloneGeneral Medicinetemporal lobe epilepsymedicine.disease030104 developmental biologychemistryNGSlcsh:Medicine (General)business030217 neurology & neurosurgeryexomemedicine.drug
researchProduct

Involvement of TRPV1 channels in the activity of the cannabinoid WIN 55,212-2 in an acute rat model of temporal lobe epilepsy

2016

The exogenous cannabinoid agonist WIN 55,212-2, (R)-(+)-[2,3-dihydro-5-methyl-3-(4-morpholinylmethyl) pyrrolo[1,2,3-de]-1,4-benzoxazin-6-Yl]-1-naphthalenylmethanone (WIN), has revealed to play a role on modulating the hyperexcitability phenomena in the hippocampus. Cannabinoid-mediated mechanisms of neuroprotection have recently been found to imply the modulation of transient receptor potential vanilloid 1 (TRPV1), a cationic channel subfamily that regulate synaptic excitation. In our study, we assessed the influence of pharmacological manipulation of TRPV1 function, alone and on WIN antiepileptic activity, in the Maximal Dentate Activation (MDA) acute model of temporal lobe epilepsy. Our r…

Male0301 basic medicineAgonistCannabinoid Receptor Modulatorsmedicine.drug_classMorpholinesmedicine.medical_treatmentTRPV1TRPV Cation ChannelsHippocampusNaphthalenesPharmacologySettore BIO/09 - FisiologiaNeuroprotection03 medical and health scienceschemistry.chemical_compound0302 clinical medicineReceptor Cannabinoid CB1Hippocampus Temporal lobe epilepsy Cannabinoids TRPV1 Capsaicin ElectrophysiologyMembrane Transport ModulatorsCannabinoid Receptor ModulatorsmedicineAnimalsRats WistarWIN 55212-2ChemistryElectric StimulationBenzoxazinesDisease Models Animal030104 developmental biologyEpilepsy Temporal LobeNeurologyAcute DiseaseAnticonvulsantslipids (amino acids peptides and proteins)Neurology (clinical)CannabinoidCapsaicinCapsazepineNeurosciencepsychological phenomena and processes030217 neurology & neurosurgerymedicine.drugEpilepsy Research
researchProduct

Role(s) of the 5-HT2C receptor in the development of maximal dentate activation in the hippocampus of anesthetized rats.

2014

Aims: Substantial evidence indicates that 5-HT2C receptors are involved in the control of neuronal network excitability and in seizure pathophysiology. Here, we have addressed the relatively unexplored relationship between temporal lobe epilepsy (TLE), the most frequent type of intractable epilepsy, and 5-HT2CRs. Methods: In this study, we investigated this issue using a model of partial complex (limbic) seizures in urethane-anesthetized rat, based on the phenomenon of maximal dentate activation (MDA) using 5-HT2C compounds, electrophysiology, immunohistochemistry, and western blotting techniques. Results: The 5-HT2C agonists mCPP (1 mg/kg, i.p) and lorcaserin (3 mg/kg, i.p), but not RO60-0…

MaleSerotonin receptorsmedicine.medical_specialtyGlutamate decarboxylaseDentate gyrus; Depression; GABA; Memory; Serotonergic2c drugs; Serotonin receptors; Temporal lobe epilepsyHippocampusHippocampal formationDentate gyruEpileptogenesisSettore BIO/09 - FisiologiaHippocampusDentate gyrus; Depression; GABA; Memory;LorcaserinRats Sprague-DawleySerotonergic 2c drugGABAEpilepsyMemoryDentate gyrus; Depression; GABA; Memory; Serotonin receptors; Temporal lobe epilepsyPhysiology (medical)Internal medicinemedicineReceptor Serotonin 5-HT2CAnimalsDentate gyrusPharmacology (medical)Serotonin receptorTemporal lobe epilepsyPharmacologyDepressionChemistryDentate gyrusDentate gyrus;Depression; GABA; Memory; Serotonergic2c drugs; Serotonin receptors; Temporal lobe epilepsyOriginal Articlesmedicine.diseaseRats5-HT2C receptorPsychiatry and Mental healthEndocrinologyDepression MentalEpilepsy Temporal LobeDentate GyrusSerotonergic2c drugAnesthetics IntravenousSerotonin 5-HT2 Receptor Agonistsmedicine.drug
researchProduct

Cannabinoid and nitric oxide signaling interplay in the modulation of hippocampal hyperexcitability: study on electrophysiological and behavioral mod…

2015

A growing bulk of evidence suggests that cannabinoid system plays a pivotal role in the control of hyperexcitability phenomena. Notwithstanding, the anticonvulsant action of cannabinoids has not been fully addressed, in particular the involvement of potential cellular neuromodulators, for instance nitric oxide. In the current study, we focused on two distinct rat models of temporal lobe epilepsy, the Maximal Dentate Activation and the pilocarpine-induced acute seizures, providing both electrophysiological and behavioral data on cannabinoid and nitrergic system interplay. We evaluated the antiepileptic effects of WIN 55,212-2, (R)-(+)-[2,3-dihydro-5-methyl-3-(4- morpholinylmethyl) pyrrolo[1,…

AgonistAM251MaleCannabinoid receptorIndazolesmedicine.drug_classmedicine.medical_treatmentMorpholinesHippocampusPharmacologyNaphthalenesNitric OxideHippocampusSettore BIO/09 - FisiologiaEpilepsyPiperidinesReceptor Cannabinoid CB1medicineAnimalshippocampus temporal lobe epilepsy cannabinoids behavior percentage of protection electrophysiology.Rats WistarWIN 55212-2Cannabinoid Receptor AgonistsDose-Response Relationship DrugCannabinoidsGeneral NeurosciencePilocarpinemedicine.diseaseEndocannabinoid systemBenzoxazinesRatsDisease Models AnimalEpilepsy Temporal LobePyrazolesCannabinoidNitric Oxide SynthasePsychologyNeurosciencemedicine.drug
researchProduct

ROLE OF NITRIC OXIDE/cGMP PATHWAY IN CANNABINOID MODULATION OF PAROXYSMAL PHENOMENA

TRPV1temporal lobe epilepsy modelnitric oxidehippocampus.cannabinoidelectrophysiologySettore BIO/09 - Fisiologiabehaviourcannabinoids; nitric oxide; TRPV1; temporal lobe epilepsy models; electrophysiology; behaviour; hippocampus.
researchProduct